A2 Psychology Depression Essays
Depression Predictions For June 2016
My depression predictions for PSYA4 and the June 2016 exam were the second most requested after Addiction when I asked our facebook study group which predictions they wanted next so here they are. Depression, like Schizophrenia is a fairly straightforward topic and there is only a handful of essays you need to learn. I should probably also mention again (incase you’re unaware) that last year the PSYA4 exam paper was in a van which was stolen a week before the exams itself – this put AQA in a predicament and they had to frantically send out a replacement paper known as PSYA4R to as many schools as they could reach – however not everyone was able to be reached in time. So last year a portion of students sat the original stolen PSYA4 paper while others who could be reached sat the replacement paper known as PSYA4R – So this has kinda screwed things up for my predictions a little as we have no idea how the replacement paper affects the pattern of questions – does it narrow down my predictions further? or does the fact that not everyone sat it mean the questions which were asked can come up again as part of the normal depression questions for this year? – Who knows…well AQA does but were going to make some big assumptions here for these predictions.
Depression Overview PSYA4
This table is taken from the specification directly and breaks down exactly the selection of questions you can be asked for the 4 different topics for section A (psychopathology as its known). The type of questions between the 4 topics are all the same and the content you learn is split between mostly explanations for the disorder and treatments (be they biological or psychological explanations or treatments).
Get A* Depression Model Essays Here
Download A* model essays for Depression HERE!
My ebook for depression covers all the possible questions you can be asked with A* grade model essays. I personally found the PSYA4 exam paper an all round easy paper – the hardest is really PSYA3 due to the sheer amount of content you are expected to know. Depression is covered in immense detail in my ebook and shows you the standard your essays need to be to hit the top banding and A* grade. If you need that extra help, want to cram for the exam last minute or want templates with A* model essays then this ebook will prove really useful for you – you can download it by clicking the image above/on the right.
My Depression Predictions For June 2016
Heres my predictions table and thoughts on what may come up below:
To access the predictions, help support Loopa by hitting the LIKE button (while logged into Facebook) to unlock the table and predictions below – if it doesn’t work straight away just hit LIKE/UNLIKE and then LIKE again while waiting a second or two between presses- sometimes it takes a few tries. You may also need to try clear your cookies and try again if it still doesn’t work.I’ll be honest and say I can’t really say how the replacement paper is going to affect PSYA4 but I’m going to give it a shot and share with you my predictions on what I would expect if I went into this exam – here goes: UPDATED TABLE and order of priority for my predictions!
- Issues surrounding the classification and diagnosis of depression – This hasn’t appeared in 3 exam windows (4 if you include the replacement paper) so I think a full essay on this is due. This is more apparent when you factor in that it’s only ever appeared as a full essay in January 2013 while other questions have made an appearance twice (except my frontrunner below). This is my second favourite to appear due to this reason and in my eyes very likely – it is also a tough question to answer and my ebook covers this with an A* grade essay. A lot of students struggled to answer these questions when it comes to reliability and validity and unless you’re writing about it in a particular way, you wont break into the top banding. It’s there if you need it.
- Biological explanations of depression – It’s been two exam windows since this appeared and my gut is telling me this is the one to prep for after my frontrunner above.
- Biological therapies for depression – Yes it appeared in the replacement paper worth 4+16 marks but my gut is telling me to prep for this (if its not my gut its that take-away I had last night). If you don’t factor in the replacement paper which not everyone sat, it’s been 6 exam windows since it appeared. I think this would be the curve ball if AQA decide to throw one so prep for this for sure.
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Psychological Theories of Depression
Saul McLeod published 2015
Depression is a mood disorder which prevents individuals from leading a normal life, at work socially or within their family. Seligman (1973) referred to depression as the ‘common cold’ of psychiatry because of its frequency of diagnosis. It is usually quite easy to see when someone is depressed.
Behaviorism emphasizes the importance of the environment in shaping behavior. The focus is on observable behavior and the conditions through which individuals' learn behavior, namely classical conditioning, operant conditioning and social learning theory. Therefore depression is the result of a person's interaction with their environment.
For example, classical conditioning proposes depression is learned through associating certain stimuli with negative emotional states. Social learning theory states behavior is learned through observation, imitation and reinforcement.
Operant conditioning states that depression is caused by the removal of positive reinforcement from the environment (Lewinsohn, 1974). Certain events, such as losing your job, induce depression because they reduce positive reinforcement from others (e.g. being around people who like you).
Depressed people usually become much less socially active. In addition depression can also be caused through inadvertent reinforcement of depressed behavior by others.
For example, when a loved one is lost, an important source of positive reinforcement has lost as well. This leads to inactivity. The main source of reinforcement is now the sympathy and attention of friends and relatives.
However this tends to reinforce maladaptive behavior i.e. weeping, complaining, talking of suicide. This eventually alienates even close friends leading to even less reinforcement, increasing social isolation and unhappiness. In other words depression is a vicious cycle in which the person is driven further and further down.
Also if the person lacks social skills or has a very rigid personality structure they may find it difficult to make the adjustments needed to look for new and alternative sources of reinforcement (Lewinsohn, 1974). So they get locked into a negative downward spiral.
Behavioral/learning theories makes sense in terms of reactive depression, where there is a clearly identifiable cause of depression. However, one of the biggest problems for the theory is that of endogenous depression. This is depression that has no apparent cause (i.e. nothing bad has happened to the person).
An additional problem of the behaviorist approach is that it fails to take into account cognitions (thoughts) influence on mood.
During the 1960's psychodynamic theories dominated psychology and psychiatry. Depression was understood in terms of:
- inwardly directed anger (Freud, 1917),
- introjection of love object loss,
- severe super-ego demands (Freud, 1917),
- excessive narcissistic, oral and/or anal personality need (Chodoff, 1972),
- loss of self-esteem (Bibring, 1953; Fenichel, 1968), and
- deprivation in the mother child relationship during the first year (Kleine, 1934).
Freud’s psychoanalytic theory is an example of the psychodynamic approach. Freud (1917) prosed that many cases of depression were due to biological factors. However, Freud also argued that some cases of depression could be linked to loss or rejection by a parent. Depression is like grief, in that it often occurs as a reaction to the loss of an important relationship.
However, there is an important difference, because depressed people regard themselves as worthless. What happens is that the individual identifies with the lost person, so that repressed anger towards the lost person is directed inwards towards the self. The inner directed anger reduces the individual’s self-esteem, and makes him/her vulnerable to experiencing depression in the future.
Freud distinguished between actual losses (e.g. death of a loved one) and symbolic losses (e.g. loss of a job). Both kinds of losses can produce depression by causing the individual to re-experience childhood episodes when they experienced loss of affection from some significant person (e.g. a parent).
Later, Freud modified his theory stating that the tendency to internalize loss objects is normal, and that depression is simply due to an excessively severe super-ego. Thus, the depressive phase occurs when the individual’s super-ego or conscience is dominant. In contrast, the manic phase occurs when the individual’s ego or rational mind asserts itself, and s/he feels control.
In order to avoid loss turning into depression, the individual needs to engage in a period of mourning work, during which s/he recalls memories of the lost one. This allows the individual to separate him/herself from the lost person, and so reduce the inner-directed anger. However, individuals very dependent on others for their sense of self-esteem may be unable to do this, and so remain extremely depressed.
Psychoanalytic theories of depression have had a profound impact on contemporary theories of depressions. For example, Beck's (1983) model of depression was influenced by psychoanalytic ideas such as the loss of self-esteem (re: Beck's negative view of self), object loss (re: the importance of loss events), external narcissistic deprivation (re: hypersensitivity to loss of social resources) and oral personality (re: sociotropic personality).
However, although being highly influential, psychoanalytic theories are difficult to test scientifically. For example, many of its central features cannot be operationally defined with sufficient precision to allow empirical investigation. Mendelson (1990) concluded his review of psychoanalytic theories of depression by stating:
'A striking feature of the impressionistic pictures of depression painted by many writers is that they have the flavor of art rather than of science and may well represent profound personal intuitions as much as they depict they raw clinical data' (p. 31).
Another criticism concerns the psychanalytic emphasis on unconscious, intrapsychic processes and early childhood experience as being limiting in that they cause clinicians to overlook additional aspects of depression. For example, conscious negative self-verbalisation (Beck, 1967), or ongoing distressing life events (Brown & Harris, 1978).
This approach focuses on people’s beliefs rather than their behavior. Depression results from systematic negative bias in thinking processes.
Emotional, behavioral (and possibly physical) symptoms result from cognitive abnormality. This means that depressed patients think differently to clinically normal people. The cognitive approach also assumes changes in thinking precede (i.e. come before) the onset of depressed mood.
Beck's (1967) Theory
One major cognitive theorist is Aaron Beck. He studied people suffering from depression and found that they appraised events in a negative way.
Beck (1967) identified three mechanisms that he thought were responsible for depression:
- The cognitive triad (of negative automatic thinking)
- Negative self schemas
- Errors in Logic (i.e. faulty information processing)
The cognitive triad are three forms of negative (i.e. helpless and critical) thinking that are typical of individuals with depression: namely negative thoughts about the self, the world and the future. These thoughts tended to be automatic in depressed people as they occurred spontaneously.
For example, depressed individuals tend to view themselves as helpless, worthless, and inadequate. They interpret events in the world in a unrealistically negative and defeatist way, and they see the world as posing obstacles that can’t be handled. Finally, they see the future as totally hopeless because their worthlessness will prevent their situation improving.
As these three components interact, they interfere with normal cognitive processing, leading to impairments in perception, memory and problem solving with the person becoming obsessed with negative thoughts.
Beck believed that depression prone individuals develop a negative self-schema. They possess a set of beliefs and expectations about themselves that are essentially negative and pessimistic. Beck claimed that negative schemas may be acquired in childhood as a result of a traumatic event. Experiences that might contribute to negative schemas include:
- Death of a parent or sibling.
- Parental rejection, criticism, overprotection, neglect or abuse.
- Bullying at school or exclusion from peer group.
However, a negative self-schema predisposes the individual to depression, and therefore someone who has acquired a cognitive triad will not necessarily develop depression. Some kind of stressful life event is required to activate this negative schema later in life. Once the negative schema are activated a number of illogical thoughts or cognitive biases seem to dominate thinking.
People with negative self schemas become prone to making logical errors in their thinking and they tend to focus selectively on certain aspects of a situation while ignoring equally relevant information.
Beck (1967) identified a number of systematic negative bias' in information processing known as logical errors or faulty thinking. These illogical thought patterns are self-defeating, and can cause great anxiety or depression for the individual. For example:
- Arbitrary Inference. Drawing a negative conclusion in the absence of supporting data.
- Selective Abstraction. Focusing on the worst aspects of any situation.
- Magnification and Minimisation. If they have a problem they make it appear bigger than it is. If they have a solution they make it smaller.
- Personalization. Negative events are interpreted as their fault.
- Dichotomous Thinking. Everything is seen as black and white. There is no in between.
Such thoughts exacerbate, and are exacerbated by the cognitive triad. Beck believed these thoughts or this way of thinking become automatic. When a person’s stream of automatic thoughts is very negative you would expect a person to become depressed. Quite often these negative thoughts will persist even in the face of contrary evidence.
Alloy et al. (1999) followed the thinking styles of young Americans in their early 20’s for 6 years. Their thinking style was tested and they were placed in either the ‘positive thinking group’ or ‘negative thinking group’. After 6 years the researchers found that only 1% of the positive group developed depression compared to 17% of the ‘negative’ group. These results indicate there may be a link between cognitive style and development of depression.
However such a study may suffer from demand characteristics. The results are also correlational. It is important to remember that the precise role of cognitive processes is yet to be determined. The maladaptive cognitions seen in depressed people may be a consequence rather than a cause of depression.
Martin Seligman (1974) proposed a cognitive explanation of depression called learned helplessness. According to Seligman’s learned helplessness theory, depression occurs when a person learns that their attempts to escape negative situations make no difference.
As a consequence they become passive and will endure aversive stimuli or environments even when escape is possible.
Seligman based his theory on research using dogs.
A dog put into a partitioned cage learns to escape when the floor is electrified. If the dog is restrained whilst being shocked it eventually stops trying to escape.
Dogs subjected to inescapable electric shocks later failed to escape from shocks even when it was possible to do so. Moreover, they exhibited some of the symptoms of depression found in humans (lethargy, sluggishness, passive in the face of stress and appetite loss).
This led Seligman (1974) to explain depression in humans in terms of learned helplessness, whereby the individual gives up trying to influence their environment because they have learned that they are helpless as a consequence of having no control over what happens to them.
Although Seligman’s account may explain depression to a certain extent, it fails to take into account cognitions (thoughts). Abramson, Seligman, and Teasdale (1978) consequently introduced a cognitive version of the theory by reformulating learned helplessness in term of attributional processes (i.e. how people explain the cause of an event).
The depression attributional style is based on three dimensions, namely locus (whether the cause is internal - to do with a person themselves, or external - to do with some aspect of the situation), stability (whether the cause is stable and permanent or unstable and transient) and global or specific (whether the cause relates to the 'whole' person or just some particular feature characteristic).
In this new version of the theory, the mere presence of a negative event was not considered sufficient to produce a helpless or depressive state. Instead, Abramson et al. argued that people who attribute failure to internal, stable, and global causes are more likely to become depressed than those who attribute failure to external, unstable and specific causes. This is because the former attributional style leads people to the conclusion that they are unable to change things for the better.
Gotlib and Colby (1987) found that people who were formerly depressed are actually no different from people who have never been depressed in terms of their tendencies to view negative events with an attitude of helpless resignation.
This suggests that helplessness could be a symptom rather than a cause of depression. Moreover, it may be that negative thinking generally is also an effect rather than a cause of depression.
Humanists believe that there are needs that are unique to the human species. According to Maslow (1962) the most important of these is the need for self-actualization (achieving out potential). The self actualizing human being has a meaningful life. Anything that blocks our striving to fulfil this need can be a cause of depression. What could cause this?
- Parents imposing conditions of worth on their children. I.e. rather than accepting the child for who s/he is and giving unconditional love, parents make love conditional on good behavior. E.g. a child may be blamed for not doing well at school, develop a negative self-image and feel depressed because of a failure to live up to parentally imposed standards.
- Some children may seek to avoid this by denying their true self and projecting an image of the kind of person they want to be. This façade or false self is an effort to please others. However the splitting off of the real self from the person you are pretending to be causes hatred of the self. The person then comes to despise themselves for living a lie.
- As adults self actualization can be undermined by unhappy relationships and unfulfilling jobs. An empty shell marriage means the person is unable to give and receive love from their partner. An alienating job means the person is denied the opportunity to be creative at work.
Abramson, L. Y., Seligman, M. E., & Teasdale, J. D. (1978). Learned helplessness in humans: critique and reformulation. Journal of abnormal psychology, 87(1), 49.
Alloy, L. B., Abramson, L. Y., Whitehouse, W. G., Hogan, M. E., Tashman, N. A., Steinberg, D. L., ... & Donovan, P. (1999). Depressogenic cognitive styles: Predictive validity, information processing and personality characteristics, and developmental origins. behavior research and therapy, 37(6), 503-531.
Beck, A. T. (1967). Depression: Causes and treatment. Philadelphia: University of Pennsylvania Press.
Beck, A. T., Epstein, N., & Harrison, R. (1983). Cognitions, attitudes and personality dimensions in depression. British Journal of Cognitive Psychotherapy.
Bibring, E. (1953). The mechanism of depression.
Brown, G. W., & Harris, T. (1978). Social origins of depression: a reply. Psychological Medicine, 8(04), 577-588.
Chodoff, P. (1972). The depressive personality: A critical review. Archives of General Psychiatry, 27(5), 666-673.
Fenichel, O. (1968). Depression and mania. The Meaning of Despair. New York: Science House.
Freud, S. (1917). Mourning and melancholia. Standard edition, 14(19), 17.
Gotlib, I. H., & Colby, C. A. (1987). Treatment of depression: An interpersonal systems approach. Pergamon Press.
Klein, M. (1934). Psychogenesis of manic-depressive states: contributions to psychoanalysis. London: Hogarth.
Lewinsohn, P. M. (1974). A behavioral approach to depression.
Maslow, A. H. (1962). Towards a psychology of being. Princeton: D. Van Nostrand Company.
Seligman, M. E. (1973). Fall into helplessness. Psychology today, 7(1), 43-48.
Seligman, M. E. (1974). Depression and learned helplessness. John Wiley & Sons.
How to reference this article:
McLeod, S. A. (2015). Psychological theories of depression. Retrieved from www.simplypsychology.org/depression.html